Associated Papers
Genetic Polymorphisms of DNA Repair Pathways in Sporadic Colorectal Carcinogenesis.
Liu J et al.
Journal of Cancer, 2019
Genetic variants in MUTYH are not associated with endometrial cancer risk.
Ashton KA et al.
Hereditary Cancer in Clinical Practice, 2009
Characterization of mutant MUTYH proteins associated with familial colorectal cancer.
Ali M et al.
Gastroenterology, 2008
Association between genetic polymorphisms of the base excision repair gene MUTYH and increased colorectal cancer risk in a Japanese population.
Tao H et al.
Cancer Science, 2008
Heterogeneous molecular mechanisms underlie attenuated familial adenomatous polyposis.
Cattaneo F et al.
Genetics in Medicine : Official Journal of the American College of Medical Genetics, 2007
Identification of MYH mutation carriers in colorectal cancer: a multicenter, case-control, population-based study.
Balaguer F et al.
Clinical Gastroenterology and Hepatology : the Official Clinical Practice Journal of the American Gastroenterological Association, 2007
Implication of MYH in colorectal polyposis.
Lefevre JH et al.
Annals of Surgery, 2006
The role of MYH and microsatellite instability in the development of sporadic colorectal cancer.
Colebatch A et al.
British Journal of Cancer, 2006
Microsatellite stable colorectal cancers in clinically suspected hereditary nonpolyposis colorectal cancer patients without vertical transmission of disease are unlikely to be caused by biallelic germline mutations in MYH.
Görgens H et al.
The Journal of Molecular Diagnostics : Jmd, 2006
Germline mutations in the MYH gene in Swedish familial and sporadic colorectal cancer.
Zhou XL et al.
Genetic Testing, 2005
A novel splice-site variant of the base excision repair gene MYH is associated with production of an aberrant mRNA transcript encoding a truncated MYH protein not localized in the nucleus.
Tao H et al.
Carcinogenesis, 2004
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