Associated Papers
MTMR regulates KRAS function by controlling plasma membrane levels of phospholipids.
Lange TE et al.
Biorxiv : the Preprint Server for Biology, 2025
STEEP mediates STING ER exit and activation of signaling.
Zhang BC et al.
Nature Immunology, 2020
32690950 (PubMed )
Supplement: EMS117976-supplement-Statistical_Source_Data_ED_Fig_6.xlsx › Fig_S6f
The PtdIns3-phosphatase MTMR3 interacts with mTORC1 and suppresses its activity.
Hao F et al.
Febs Letters, 2016
Myotubularin-related proteins 3 and 4 interact with polo-like kinase 1 and centrosomal protein of 55 kDa to ensure proper abscission.
St-Denis N et al.
Molecular & Cellular Proteomics : Mcp, 2015
PtdIns(3)P-bound UVRAG coordinates Golgi-ER retrograde and Atg9 transport by differential interactions with the ER tether and the beclin 1 complex.
He S et al.
Nature Cell Biology, 2013
The PTEN and Myotubularin phosphoinositide 3-phosphatases: linking lipid signalling to human disease.
Davies EM et al.
Sub-cellular Biochemistry, 2012
Charcot-Marie-Tooth type 4B demyelinating neuropathy: deciphering the role of MTMR phosphatases.
Previtali SC et al.
Expert Reviews in Molecular Medicine, 2007
Myotubularin phosphatases: policing 3-phosphoinositides.
Robinson FL et al.
Trends in Cell Biology, 2006
Systematic analysis of myotubularins: heteromeric interactions, subcellular localisation and endosome related functions.
Lorenzo O et al.
Journal of Cell Science, 2006
The myotubularin family of lipid phosphatases.
Clague MJ et al.
Traffic (copenhagen, Denmark), 2005
Analysis of phosphoinositide binding domain properties within the myotubularin-related protein MTMR3.
Lorenzo O et al.
Journal of Cell Science, 2005
Characterization of MTMR3. an inositol lipid 3-phosphatase with novel substrate specificity.
Walker DM et al.
Current Biology : Cb, 2001
Select a paper to view details