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A chemical genetic screen identifies Aurora kinases as a therapeutic target in EGFR T790M negative, gefitinib-resistant head and neck squamous cell carcinoma (HNSCC).
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Identification of NCAN as a candidate gene for developmental dyslexia.
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Co-existence of BRAF and NRAS driver mutations in the same melanoma cells results in heterogeneity of targeted therapy resistance.
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Oncotarget, 2016
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A novel null homozygous mutation confirms CACNA2D2 as a gene mutated in epileptic encephalopathy.
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Plos One, 2013
24358150 (PubMed )
Supplement: pone.0082154.s002.xls › Sheet1
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