Associated Papers
Oxidative DNA damage induces hypomethylation in a compromised base excision repair colorectal tumourigenesis.
Furlan D et al.
British Journal of Cancer, 2017
28141798 (PubMed )
Supplement: bjc20179x1.pdf
Increased risk for colorectal adenomas and cancer in mono-allelic MUTYH mutation carriers: results from a cohort of North-African Jews.
Rosner G et al.
Familial Cancer, 2015
Identification of patients with (atypical) MUTYH-associated polyposis by KRAS2 c.34G > T prescreening followed by MUTYH hotspot analysis in formalin-fixed paraffin-embedded tissue.
van Puijenbroek M et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research, 2008
Base excision repair and the role of MUTYH.
Kairupan C et al.
Hereditary Cancer in Clinical Practice, 2007
Heterogeneous molecular mechanisms underlie attenuated familial adenomatous polyposis.
Cattaneo F et al.
Genetics in Medicine : Official Journal of the American College of Medical Genetics, 2007
MUTYH-associated polyposis--from defect in base excision repair to clinical genetic testing.
Cheadle JP et al.
Dna Repair, 2007
[Clinical features and current progress of familial adenomatous polyposis].
Ushio K
Nihon Rinsho. Japanese Journal of Clinical Medicine, 2000
Mechanism of oxidative DNA damage repair and relevance to human pathology.
D'Errico M et al.
Mutation Research, 0
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