Associated Papers
Microhomology-mediated end joining drives complex rearrangements and overexpression of MYC and PVT1 in multiple myeloma.
Mikulasova A et al.
Haematologica, 2020
A common functional consequence of tumor-derived mutations within c-MYC.
Chakraborty AA et al.
Oncogene, 2015
24998853 (PubMed )
Supplement: NIHMS593237-supplement-7.pdf
Quantifying the role of aberrant somatic hypermutation in transformation of follicular lymphoma.
Halldórsdóttir AM et al.
Leukemia Research, 2008
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