Associated Papers
Case Report: Novel variants in the MYD88 gene (c.104T>C, c.141G>C) in a patient with recurrent abscesses as a cause of type 68 immunodeficiency.
Buianova AA et al.
Frontiers in Immunology, 2025
Three Unrelated Patients of Roma Ethnicity from a Single Center Carrying the Same Deletion in MYD88 Gene: A Founder Effect?
Romano R et al.
Life (basel, Switzerland), 2024
Humans with inherited MyD88 and IRAK-4 deficiencies are predisposed to hypoxemic COVID-19 pneumonia.
García-García A et al.
The Journal of Experimental Medicine, 2023
A Novel Kindred with MyD88 Deficiency.
Bucciol G et al.
Journal of Clinical Immunology, 2022
Toll-Like Receptor Signaling in the Establishment and Function of the Immune System.
Aluri J et al.
Cells, 2021
Modeling MyD88 Deficiency In Vitro Provides New Insights in Its Function.
Craig-Mueller N et al.
Frontiers in Immunology, 2020
Targeted next-generation sequencing revealed MYD88 deficiency in a child with chronic yersiniosis and granulomatous lymphadenitis.
Giardino G et al.
The Journal of Allergy and Clinical Immunology, 2016
Rare mendelian primary immunodeficiency diseases associated with impaired NF-κB signaling.
Paciolla M et al.
Genes and Immunity, 2015
Two human MYD88 variants, S34Y and R98C, interfere with MyD88-IRAK4-myddosome assembly.
George J et al.
The Journal of Biological Chemistry, 2011
The transmembrane activator TACI triggers immunoglobulin class switching by activating B cells through the adaptor MyD88.
He B et al.
Nature Immunology, 2010
Pyogenic bacterial infections in humans with MyD88 deficiency.
von Bernuth H et al.
Science (new York, N.y.), 2008
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