Associated Papers
Case Report: Novel variants in the MYD88 gene (c.104T>C, c.141G>C) in a patient with recurrent abscesses as a cause of type 68 immunodeficiency.
Buianova AA et al.
Frontiers in Immunology, 2025
Toll-Like Receptor Signaling in the Establishment and Function of the Immune System.
Aluri J et al.
Cells, 2021
Rare mendelian primary immunodeficiency diseases associated with impaired NF-κB signaling.
Paciolla M et al.
Genes and Immunity, 2015
Genetic variation in Toll-like receptors and disease susceptibility.
Netea MG et al.
Nature Immunology, 2012
Two human MYD88 variants, S34Y and R98C, interfere with MyD88-IRAK4-myddosome assembly.
George J et al.
The Journal of Biological Chemistry, 2011
The transmembrane activator TACI triggers immunoglobulin class switching by activating B cells through the adaptor MyD88.
He B et al.
Nature Immunology, 2010
The role of Toll-like receptor signaling in human immunodeficiencies.
Suhir H et al.
Clinical Reviews in Allergy & Immunology, 2010
IRAK-4- and MyD88-dependent pathways are essential for the removal of developing autoreactive B cells in humans.
Isnardi I et al.
Immunity, 2008
Pyogenic bacterial infections in humans with MyD88 deficiency.
von Bernuth H et al.
Science (new York, N.y.), 2008
Mutations in STAT3 and IL12RB1 impair the development of human IL-17-producing T cells.
de Beaucoudrey L et al.
The Journal of Experimental Medicine, 2008
18591412 (PubMed )
Supplement: jem.20080321_5.pdf
Select a paper to view details