Associated Papers
Gene Therapy for Inherited Cardiac Arrhythmias.
Turkieltaub Paredes MR et al.
Journal of Cardiovascular Translational Research, 2025
Mechanisms Underlying Sinus Node Dysfunction in a Rat Model of Genetic Atrial Cardiomyopathy.
Marcoux E et al.
Circulation. Arrhythmia and Electrophysiology, 2025
Genetic Atrial Cardiomyopathies: Common Features, Specific Differences, and Broader Relevance to Understanding Atrial Cardiomyopathy.
Marcoux E et al.
Circulation. Arrhythmia and Electrophysiology, 2023
Myosin light-chain 4 gene-transfer attenuates atrial fibrosis while correcting autophagic flux dysregulation.
Zhong Y et al.
Redox Biology, 2023
Genetic variants, pathophysiological pathways, and oral anticoagulation in patients with hypertrophic cardiomyopathy and atrial fibrillation.
Wang S et al.
Frontiers in Cardiovascular Medicine, 2023
Long-term amelioration of an early-onset familial atrial fibrillation model with AAV-mediated in vivo gene therapy.
Hu H et al.
Fundamental Research, 2022
Cytoskeletal Protein Variants Driving Atrial Fibrillation: Potential Mechanisms of Action.
van Wijk SW et al.
Cells, 2022
Modeling Human Cardiac Arrhythmias: Insights from Zebrafish.
Gauvrit S et al.
Journal of Cardiovascular Development and Disease, 2022
The molecular genetic basis of atrial fibrillation.
Huang X et al.
Human Genetics, 2020
Expanding the Clinical Phenotype of Emerinopathies: Atrial Standstill and Left Ventricular Noncompaction.
Gollob MH
Circulation. Arrhythmia and Electrophysiology, 2020
Rs4968309 in Myosin Light Chain 4 (MYL4) Associated With Atrial Fibrillation Onset and Predicts Clinical Outcomes After Catheter Ablation in Atrial Fibrillation Patients Without Structural Heart Disease.
Zhong Y et al.
Circulation Journal : Official Journal of the Japanese Circulation Society, 2019
Dysfunction of Myosin Light-Chain 4 (MYL4) Leads to Heritable Atrial Cardiomyopathy With Electrical, Contractile, and Structural Components: Evidence From Genetically-Engineered Rats.
Peng W et al.
Journal of the American Heart Association, 2017
A mutation in the atrial-specific myosin light chain gene (MYL4) causes familial atrial fibrillation.
Orr N et al.
Nature Communications, 2016
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