Associated Papers
Genetic rodent models of amyotrophic lateral sclerosis.
Van Den Bosch L
Journal of Biomedicine & Biotechnology, 2011
Mutant copper-zinc superoxide dismutase associated with amyotrophic lateral sclerosis binds to adenine/uridine-rich stability elements in the vascular endothelial growth factor 3'-untranslated region.
Li X et al.
Journal of Neurochemistry, 2009
Disease mechanisms revealed by transcription profiling in SOD1-G93A transgenic mouse spinal cord.
Olsen MK et al.
Annals of Neurology, 2001
Superoxide dismutase-1 mutation-related neurotoxicity in familial amyotrophic lateral sclerosis.
Shibata N et al.
Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders : Official Publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases, 2000
Overexpression of neurofilament subunit NF-L and NF-H extends survival of a mouse model for amyotrophic lateral sclerosis.
Kong J et al.
Neuroscience Letters, 2000
Oxidative stress and motor neurone disease.
Cookson MR et al.
Brain Pathology (zurich, Switzerland), 1999
Neurofilaments and orthograde transport are reduced in ventral root axons of transgenic mice that express human SOD1 with a G93A mutation.
Zhang B et al.
The Journal of Cell Biology, 1997
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