Associated Papers
Increased burden of rare risk variants across gene expression networks predisposes to sporadic Parkinson's disease.
Eubanks E et al.
Cell Reports, 2025
Mouse α-synuclein fibrils are structurally and functionally distinct from human fibrils associated with Lewy body diseases.
Sokratian A et al.
Science Advances, 2024
39485845 (PubMed )
Supplement: sciadv.adq3539_sm.pdf
Autonomic dysfunction in genetic forms of synucleinopathies.
Chelban V et al.
Movement Disorders : Official Journal of the Movement Disorder Society, 2018
Autosomal dominant Parkinson's disease and the route to new therapies.
Morris HR
Expert Review of Neurotherapeutics, 2007
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