Associated Papers
Restoration of Lysosomal Hydrolase Activities by LRRK2 Inhibition in GBA1- and LRRK2-Associated Parkinson's Disease Patient-Derived Cells.
Usenko TS et al.
Journal of Neurochemistry, 2025
Autophagy Process in Parkinson's Disease Depends on Mutations in the GBA1 and LRRK2 Genes.
Bezrukova AI et al.
Biochemical Genetics, 2025
Glitazone Treatment Rescues Phenotypic Deficits in a Fly Model of Gaucher/Parkinson's Disease.
Shola-Dare O et al.
International Journal of Molecular Sciences, 2021
Modelling Parkinson's Disease: iPSCs towards Better Understanding of Human Pathology.
Avazzadeh S et al.
Brain Sciences, 2021
GBA Mutations Influence the Release and Pathological Effects of Small Extracellular Vesicles from Fibroblasts of Patients with Parkinson's Disease.
Cerri S et al.
International Journal of Molecular Sciences, 2021
Genetic predispositions of Parkinson's disease revealed in patient-derived brain cells.
Tran J et al.
Npj Parkinson's Disease, 2020
Mitochondrial dysfunction and mitophagy defect triggered by heterozygous GBA mutations.
Li H et al.
Autophagy, 2019
α-Synuclein accumulation and GBA deficiency due to L444P GBA mutation contributes to MPTP-induced parkinsonism.
Yun SP et al.
Molecular Neurodegeneration, 2018
29310663 (PubMed )
Supplement: 13024_2017_233_MOESM9_ESM.pdf
Mitochondria: A Common Target for Genetic Mutations and Environmental Toxicants in Parkinson's Disease.
Helley MP et al.
Frontiers in Genetics, 2017
Ambroxol improves lysosomal biochemistry in glucocerebrosidase mutation-linked Parkinson disease cells.
McNeill A et al.
Brain : a Journal of Neurology, 2014
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