Associated Papers
OAS1 and OAS3 genetic variants enhance inflammatory responses to SARS-CoV-2.
DeDiego ML et al.
Iscience, 2025
41438061 (PubMed )
Supplement: mmc3.xlsx › Table_S1
Germline predisposition in multiple myeloma.
Martins Rodrigues F et al.
Iscience, 2025
39845416 (PubMed )
Supplement: mmc3.xlsx › ST2D.Vars_in_all_genes_MMRF
The role of inflammatory gene polymorphisms in severe COVID-19: a review.
Yip JQ et al.
Virology Journal, 2024
Individual genetic variability mainly of Proinflammatory cytokines, cytokine receptors, and toll-like receptors dictates pathophysiology of COVID-19 disease.
Vakil MK et al.
Journal of Medical Virology, 2022
An overview of human proteins and genes involved in SARS-CoV-2 infection.
Jahanafrooz Z et al.
Gene, 2022
Human genetic basis of severe or critical illness in COVID-19.
Ji XS et al.
Frontiers in Cellular and Infection Microbiology, 2022
Functional prediction and comparative population analysis of variants in genes for proteases and innate immunity related to SARS-CoV-2 infection.
Klaassen K et al.
Infection, Genetics and Evolution : Journal of Molecular Epidemiology and Evolutionary Genetics in Infectious Diseases, 2020
Recurrent Loss-of-Function Mutations Reveal Costs to OAS1 Antiviral Activity in Primates.
Carey CM et al.
Cell Host & Microbe, 2019
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