Associated Papers
OAS1 and OAS3 genetic variants enhance inflammatory responses to SARS-CoV-2.
DeDiego ML et al.
Iscience, 2025
Large-scale genome sequencing redefines the genetic footprints of high-altitude adaptation in Tibetans.
Zheng W et al.
Genome Biology, 2023
Genetic regulation of OAS1 nonsense-mediated decay underlies association with COVID-19 hospitalization in patients of European and African ancestries.
Banday AR et al.
Nature Genetics, 2022
35835913 (PubMed )
Supplement: 41588_2022_1113_MOESM4_ESM.xlsx › Table_S1
Common, low-frequency, rare, and ultra-rare coding variants contribute to COVID-19 severity.
Fallerini C et al.
Human Genetics, 2022
34889978 (PubMed )
Supplement: 439_2021_2397_MOESM19_ESM.xlsx › Foglio1
Individualized Proteogenomics Reveals the Mutational Landscape of Melanoma Patients in Response to Immunotherapy.
Schmitt M et al.
Cancers, 2021
34771574 (PubMed )
Supplement: cancers-13-05411-s001.zip › cancers-1421686_Table_S4.xlsx › Sheet_1
Genetic regulation of OAS1 nonsense-mediated decay underlies association with risk of severe COVID-19.
Banday AR et al.
Medrxiv : the Preprint Server for Health Sciences, 2021
34282422 (PubMed )
Supplement: media-1.xlsx › Table_S1
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