Associated Papers
OAS1 and OAS3 genetic variants enhance inflammatory responses to SARS-CoV-2.
DeDiego ML et al.
Iscience, 2025
Inborn errors of OAS-RNase L in SARS-CoV-2-related multisystem inflammatory syndrome in children.
Lee D et al.
Science (new York, N.y.), 2023
Genetic regulation of OAS1 nonsense-mediated decay underlies association with risk of severe COVID-19.
Banday AR et al.
Medrxiv : the Preprint Server for Health Sciences, 2021
34282422 (PubMed )
Supplement: media-1.xlsx › Table_S1
Genetic architecture differences between pediatric and adult-onset inflammatory bowel diseases in the Polish population.
Ostrowski J et al.
Scientific Reports, 2016
28008999 (PubMed )
Supplement: srep39831-s2.xls › Table_S7
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