Associated Papers
Variation in the mu-opioid receptor gene (OPRM1) moderates the influence of maternal sensitivity on child attachment.
Tchalova K et al.
Translational Psychiatry, 2024
Genetic and epigenetic effects on couple adjustment in context of romantic relationship: A scoping systematic review.
Khani P et al.
Frontiers in Genetics, 2023
Genetic polymorphisms in the serotonin, dopamine and opioid pathways influence social attention in rhesus macaques (Macaca mulatta).
Howarth ERI et al.
Plos One, 2023
Addiction associated N40D mu-opioid receptor variant modulates synaptic function in human neurons.
Halikere A et al.
Molecular Psychiatry, 2020
Parental overcontrol x OPRM1 genotype interaction predicts school-aged children's sympathetic nervous system activation in response to performance challenge.
Partington LC et al.
Research in Developmental Disabilities, 2018
Non-invasive genotyping with a massively parallel sequencing panel for the detection of SNPs in HPA-axis genes.
Gutleb DR et al.
Scientific Reports, 2018
OPRM1 genotype interacts with serotonin system dysfunction to predict alcohol-heightened aggression in primates.
Driscoll CA et al.
Addiction Biology, 2017
Convergent Balancing Selection on the Mu-Opioid Receptor in Primates.
Sweeney CG et al.
Molecular Biology and Evolution, 2017
Oxytocin and Opioid Receptor Gene Polymorphisms Associated with Greeting Behavior in Dogs.
Kubinyi E et al.
Frontiers in Psychology, 2017
Opioid Antagonists and the A118G Polymorphism in the μ-Opioid Receptor Gene: Effects of GSK1521498 and Naltrexone in Healthy Drinkers Stratified by OPRM1 Genotype.
Ziauddeen H et al.
Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology, 2016
Mouse model of OPRM1 (A118G) polymorphism increases sociability and dominance and confers resilience to social defeat.
Briand LA et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 2015
Pharmacogenetic approaches in the treatment of alcohol use disorders: addressing clinical utility and implementation thresholds.
Hendershot CS
Addiction Science & Clinical Practice, 2014
Large-scale polymorphism discovery in macaque G-protein coupled receptors.
Goswami DB et al.
Bmc Genomics, 2013
Non-human primate models of alcohol-related phenotypes: the influence of genetic and environmental factors.
Barr CS
Current Topics in Behavioral Neurosciences, 2013
Consequences of the 118A>G polymorphism in the OPRM1 gene: translation from bench to bedside?
Mura E et al.
Journal of Pain Research, 2013
Genetic load is associated with hypothalamic-pituitary-adrenal axis dysregulation in macaques.
Ferguson B et al.
Genes, Brain, and Behavior, 2012
Testing multiple levels of influence in the intergenerational transmission of alcohol disorders from a developmental perspective: the example of alcohol use promoting peers and μ-opioid receptor M1 variation.
Chassin L et al.
Development and Psychopathology, 2012
The genetics of the opioid system and specific drug addictions.
Levran O et al.
Human Genetics, 2012
The role of the Asn40Asp polymorphism of the mu opioid receptor gene (OPRM1) on alcoholism etiology and treatment: a critical review.
Ray LA et al.
Alcoholism, Clinical and Experimental Research, 2012
Influence of the OPRM1 gene polymorphism upon children's degree of withdrawal and brain activation in response to facial expressions.
Bertoletti E et al.
Developmental Cognitive Neuroscience, 2012
OPRM1 gene variation influences hypothalamic-pituitary-adrenal axis function in response to a variety of stressors in rhesus macaques.
Schwandt ML et al.
Psychoneuroendocrinology, 2011
Mu-opioid receptor (OPRM1) variation, oxytocin levels and maternal attachment in free-ranging rhesus macaques Macaca mulatta.
Higham JP et al.
Behavioral Neuroscience, 2011
Social hedonic capacity is associated with the A118G polymorphism of the mu-opioid receptor gene (OPRM1) in adult healthy volunteers and psychiatric patients.
Troisi A et al.
Social Neuroscience, 2011
A pharmacogenetic model of naltrexone-induced attenuation of alcohol consumption in rhesus monkeys.
Vallender EJ et al.
Drug and Alcohol Dependence, 2010
OPRM1 SNP (A118G): involvement in disease development, treatment response, and animal models.
Mague SD et al.
Drug and Alcohol Dependence, 2010
Suppression of alcohol preference by naltrexone in the rhesus macaque: a critical role of genetic variation at the micro-opioid receptor gene locus.
Barr CS et al.
Biological Psychiatry, 2010
Strategies for performing genotype-phenotype association studies in nonhuman primates.
Barr CS
Methods (san Diego, Calif.), 2009
Rhesus monkey tryptophan hydroxylase-2 coding region haplotypes affect mRNA stability.
Chen GL et al.
Neuroscience, 2008
CRH haplotype as a factor influencing cerebrospinal fluid levels of corticotropin-releasing hormone, hypothalamic-pituitary-adrenal axis activity, temperament, and alcohol consumption in rhesus macaques.
Barr CS et al.
Archives of General Psychiatry, 2008
Human expression variation in the mu-opioid receptor is paralleled in rhesus macaque.
Vallender EJ et al.
Behavior Genetics, 2008
Variation at the mu-opioid receptor gene (OPRM1) influences attachment behavior in infant primates.
Barr CS et al.
Proceedings of the National Academy of Sciences of the United States of America, 2008
A functional polymorphism of the micro-opioid receptor gene is associated with completed suicides.
Hishimoto A et al.
Journal of Neural Transmission (vienna, Austria : 1996), 2008
Pharmacogenetic treatments for drug addiction: alcohol and opiates.
Haile CN et al.
The American Journal of Drug and Alcohol Abuse, 2008
A mu-opioid receptor single nucleotide polymorphism in rhesus monkey: association with stress response and aggression.
Miller GM et al.
Molecular Psychiatry, 2004
μ-Opioid receptor gene (OPRM1) polymorphism A118G: lack of association in Finnish populations with alcohol dependence or alcohol consumption.
Rouvinen-Lagerström N et al.
Alcohol and Alcoholism (oxford, Oxfordshire), 0
Identification of genetic variations in μ opioid receptor in cats.
Sasaki K et al.
Molecular Pain, 0