Associated Papers
Genetic-environmental Risk Factors for Tooth Agenesis: A Familial Case-control Study in China.
Guo X et al.
International Dental Journal, 2025
An inclusive study of deleterious missense PAX9 variants using user-friendly tools reveals structural, functional alterations, as well as potential therapeutic targets.
Ranjan P et al.
International Journal of Biological Macromolecules, 2023
The clinical significance and correlative signaling pathways of paired box gene 9 in development and carcinogenesis.
Bhol CS et al.
Biochimica Et Biophysica Acta. Reviews on Cancer, 2021
MicroRNA-655-3p and microRNA-497-5p inhibit cell proliferation in cultured human lip cells through the regulation of genes related to human cleft lip.
Gajera M et al.
Bmc Medical Genomics, 2019
31122291 (PubMed )
Supplement: 12920_2019_535_MOESM1_ESM.zip › 4_Human_Cleft_Lip__Table_S4__Significant_R2.xlsx › Multiple_Genes_Significant
Genetic analysis: Wnt and other pathways in nonsyndromic tooth agenesis.
Yu M et al.
Oral Diseases, 2019
Genotyping analysis of the Pax9 Gene in patients with maxillary canine impaction.
Vitria EE et al.
F1000research, 2019
Evaluating rare coding variants as contributing causes to non-syndromic cleft lip and palate.
Leslie EJ et al.
Clinical Genetics, 2013
Association between PAX9 single-nucleotide polymorphisms and nonsyndromic cleft lip with or without cleft palate.
Lee JK et al.
The Journal of Craniofacial Surgery, 2012
PAX9 and TGFB3 are linked to susceptibility to nonsyndromic cleft lip with or without cleft palate in the Japanese: population-based and family-based candidate gene analyses.
Ichikawa E et al.
Journal of Human Genetics, 2006
Select a paper to view details