Associated Papers
Genetic studies in human prion diseases.
Jeong BH et al.
Journal of Korean Medical Science, 2014
Polymorphic microsatellite sites in the PRNP region point to excess of homozygotes in Creutzfeldt-Jakob disease patients.
Geldermann H et al.
Gene, 2006
Polymorphisms at codons 56 and 174 of the prion-like protein gene (PRND) are not associated with sporadic Creutzfeldt-Jakob disease.
Jeong BH et al.
Journal of Human Genetics, 2005
Polymorphisms in the prion protein gene and in the doppel gene increase susceptibility for Creutzfeldt-Jakob disease.
Croes EA et al.
European Journal of Human Genetics : Ejhg, 2004
The human "prion-like" protein Doppel is expressed in both Sertoli cells and spermatozoa.
Peoc'h K et al.
The Journal of Biological Chemistry, 2002
Sporadic--but not variant--Creutzfeldt-Jakob disease is associated with polymorphisms upstream of PRNP exon 1.
Mead S et al.
American Journal of Human Genetics, 2001
Polymorphisms within the prion-like protein gene (Prnd) and their implications in human prion diseases, Alzheimer's disease and other neurological disorders.
Schröder B et al.
Human Genetics, 2001
First report of polymorphisms in the prion-like protein gene (PRND): implications for human prion diseases.
Peoc'h K et al.
Neuroscience Letters, 2000
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