Associated Papers
Computational Search for Inhibitors of SOD1 Mutant Infectivity as Potential Therapeutics for ALS Disease.
Carnaroli M et al.
International Journal of Molecular Sciences, 2025
Amyotrophic Lateral Sclerosis Genes in Drosophila melanogaster.
Layalle S et al.
International Journal of Molecular Sciences, 2021
Preserving neuromuscular synapses in ALS by stimulating MuSK with a therapeutic agonist antibody.
Cantor S et al.
Elife, 2018
Expression of wild-type human superoxide dismutase-1 in mice causes amyotrophic lateral sclerosis.
Graffmo KS et al.
Human Molecular Genetics, 2013
SOD1 and DJ-1 converge at Nrf2 pathway: a clue for antioxidant therapeutic potential in neurodegeneration.
Milani P et al.
Oxidative Medicine and Cellular Longevity, 2013
Sorting of the yeast vacuolar-type, proton-translocating ATPase enzyme complex (V-ATPase): identification of a necessary and sufficient Golgi/endosomal retention signal in Stv1p.
Finnigan GC et al.
The Journal of Biological Chemistry, 2012
Misfolded superoxide dismutase-1 in CSF from amyotrophic lateral sclerosis patients.
Zetterström P et al.
Journal of Neurochemistry, 2011
Dorfin ameliorates phenotypes in a transgenic mouse model of amyotrophic lateral sclerosis.
Sone J et al.
Journal of Neuroscience Research, 2010
Proteomic characterization of lipid raft proteins in amyotrophic lateral sclerosis mouse spinal cord.
Zhai J et al.
The Febs Journal, 2009
Destabilizing protein polymorphisms in the genetic background direct phenotypic expression of mutant SOD1 toxicity.
Gidalevitz T et al.
Plos Genetics, 2009
Genetic polymorphisms of selected DNA repair genes, estrogen and progesterone receptor status, and breast cancer risk.
Lee KM et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research, 2005
Residues of VP26 of herpes simplex virus type 1 that are required for its interaction with capsids.
Desai P et al.
Journal of Virology, 2003
Identification of functionally important residues of the epidermal growth factor-2 domain of factor IX by alanine-scanning mutagenesis. Residues Asn(89)-Gly(93) are critical for binding factor VIIIa.
Chang YJ et al.
The Journal of Biological Chemistry, 2002
Neuron-specific expression of mutant superoxide dismutase 1 in transgenic mice does not lead to motor impairment.
Pramatarova A et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 2001