Associated Papers
Compound heterozygous IFT81 variations in a skeletal ciliopathy patient cause Bardet-Biedl syndrome-like ciliary defects.
Tasaki K et al.
Human Molecular Genetics, 2023
CEP19-RABL2-IFT-B axis controls BBSome-mediated ciliary GPCR export.
Zhou Z et al.
Molecular Biology of the Cell, 2022
Rabl2 GTP hydrolysis licenses BBSome-mediated export to fine-tune ciliary signaling.
Duan S et al.
The Embo Journal, 2021
RABL2 positively controls localization of GPCRs in mammalian primary cilia.
Dateyama I et al.
Journal of Cell Science, 2019
The CEP19-RABL2 GTPase Complex Binds IFT-B to Initiate Intraflagellar Transport at the Ciliary Base.
Kanie T et al.
Developmental Cell, 2017
RABL2 interacts with the intraflagellar transport-B complex and CEP19 and participates in ciliary assembly.
Nishijima Y et al.
Molecular Biology of the Cell, 2017
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