Associated Papers
MOG1L18F-mediated increase in late sodium current produces Long QT Syndrome.
Socuéllamos PG et al.
Medrxiv : the Preprint Server for Health Sciences, 2025
Molecular mechanism for the interaction of MOG1 with the intracellular loop II of cardiac sodium channel Nav1.5 and its role in arrhythmias.
Bai X et al.
Journal of Molecular and Cellular Cardiology, 2025
Inherited and Acquired Rhythm Disturbances in Sick Sinus Syndrome, Brugada Syndrome, and Atrial Fibrillation: Lessons from Preclinical Modeling.
Iop L et al.
Cells, 2021
Genetic interpretation and clinical translation of minor genes related to Brugada syndrome.
Campuzano O et al.
Human Mutation, 2019
Mechanistic insights into the interaction of the MOG1 protein with the cardiac sodium channel Nav1.5 clarify the molecular basis of Brugada syndrome.
Yu G et al.
The Journal of Biological Chemistry, 2018
Cardiac channelopathies: The role of sodium channel mutations.
Fonseca DJ et al.
Revista Portuguesa De Cardiologia, 2018
Brugada syndrome: a fatal disease with complex genetic etiologies - still a long way to go.
Wu Y et al.
Forensic Sciences Research, 2017
Cardiac sodium channel regulator MOG1 regulates cardiac morphogenesis and rhythm.
Zhou J et al.
Scientific Reports, 2016
Cardiac Sodium Channel Mutations: Why so Many Phenotypes?
Liu M et al.
Current Topics in Membranes, 2016
Protein assemblies of sodium and inward rectifier potassium channels control cardiac excitability and arrhythmogenesis.
Willis BC et al.
American Journal of Physiology. Heart and Circulatory Physiology, 2015
Diseases caused by mutations in Nav1.5 interacting proteins.
Kyle JW et al.
Cardiac Electrophysiology Clinics, 2014
Cardiac sodium channel mutations: why so many phenotypes?
Liu M et al.
Nature Reviews. Cardiology, 2014
Identifying potential functional impact of mutations and polymorphisms: linking heart failure, increased risk of arrhythmias and sudden cardiac death.
Jagu B et al.
Frontiers in Physiology, 2013
Arrhythmogenesis in Brugada syndrome: impact and constrains of current concepts.
Bébarová M
International Journal of Cardiology, 2013
Channelopathies from mutations in the cardiac sodium channel protein complex.
Adsit GS et al.
Journal of Molecular and Cellular Cardiology, 2013
MOG1 rescues defective trafficking of Na(v)1.5 mutations in Brugada syndrome and sick sinus syndrome.
Chakrabarti S et al.
Circulation. Arrhythmia and Electrophysiology, 2013
MOG1: a new susceptibility gene for Brugada syndrome.
Kattygnarath D et al.
Circulation. Cardiovascular Genetics, 2011
Sodium current and potassium transient outward current genes in Brugada syndrome: screening and bioinformatics.
Holst AG et al.
The Canadian Journal of Cardiology, 0