Associated Papers
BRCA1 and 53BP1 regulate reprogramming efficiency by mediating DNA repair pathway choice at replication-associated double-strand breaks.
Georgieva D et al.
Cell Reports, 2024
ATM/ATR Phosphorylation of CtIP on Its Conserved Sae2-like Domain Is Required for Genotoxin-Induced DNA Resection but Dispensable for Animal Development.
Wu-Baer F et al.
Cells, 2023
Wwox Binding to the Murine Brca1-BRCT Domain Regulates Timing of Brip1 and CtIP Phospho-Protein Interactions with This Domain at DNA Double-Strand Breaks, and Repair Pathway Choice.
Park D et al.
International Journal of Molecular Sciences, 2022
And-1 coordinates with CtIP for efficient homologous recombination and DNA damage checkpoint maintenance.
Chen Y et al.
Nucleic Acids Research, 2017
MRN, CtIP, and BRCA1 mediate repair of topoisomerase II-DNA adducts.
Aparicio T et al.
The Journal of Cell Biology, 2016
BRCA1, a 'complex' protein involved in the maintenance of genomic stability.
Savage KI et al.
The Febs Journal, 2015
CtIP-mediated resection is essential for viability and can operate independently of BRCA1.
Polato F et al.
The Journal of Experimental Medicine, 2014
The interaction between CtIP and BRCA1 is not essential for resection-mediated DNA repair or tumor suppression.
Reczek CR et al.
The Journal of Cell Biology, 2013
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