Associated Papers
DNA-PKcs-dependent phosphorylation of RECQL4 promotes NHEJ by stabilizing the NHEJ machinery at DNA double-strand breaks.
Lu H et al.
Nucleic Acids Research, 2022
RECQ DNA Helicases and Osteosarcoma.
Lu L et al.
Advances in Experimental Medicine and Biology, 2020
ATP-dependent helicase activity is dispensable for the physiological functions of Recql4.
Castillo-Tandazo W et al.
Plos Genetics, 2019
Chromosome alignment maintenance requires the MAP RECQL4, mutated in the Rothmund-Thomson syndrome.
Yokoyama H et al.
Life Science Alliance, 2019
RecQL4-Aurora B kinase axis is essential for cellular proliferation, cell cycle progression, and mitotic integrity.
Fang H et al.
Oncogenesis, 2018
Senescence induced by RECQL4 dysfunction contributes to Rothmund-Thomson syndrome features in mice.
Lu H et al.
Cell Death & Disease, 2014
The RECQL4 protein, deficient in Rothmund-Thomson syndrome is active on telomeric D-loops containing DNA metabolism blocking lesions.
Ferrarelli LK et al.
Dna Repair, 2013
RECQL4 in genomic instability and aging.
Croteau DL et al.
Trends in Genetics : Tig, 2012
RAPADILINO RECQL4 mutant protein lacks helicase and ATPase activity.
Croteau DL et al.
Biochimica Et Biophysica Acta, 2012
The human RecQ helicases BLM and RECQL4 cooperate to preserve genome stability.
Singh DK et al.
Nucleic Acids Research, 2012
Conserved helicase domain of human RecQ4 is required for strand annealing-independent DNA unwinding.
Rossi ML et al.
Dna Repair, 2010
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