Associated Papers
Distinct roles of RECQL5 in RAD51-mediated fork reversal and transcription elongation.
Nagraj T et al.
Nucleic Acids Research, 2025
Genome-scale mapping of DNA damage suppressors through phenotypic CRISPR-Cas9 screens.
Zhao Y et al.
Molecular Cell, 2023
RECQ5 Helicase Cooperates with MUS81 Endonuclease in Processing Stalled Replication Forks at Common Fragile Sites during Mitosis.
Di Marco S et al.
Molecular Cell, 2017
Human RECQ Helicase Pathogenic Variants, Population Variation and "Missing" Diseases.
Fu W et al.
Human Mutation, 2017
Altered RECQL5 expression in urothelial bladder carcinoma increases cellular proliferation and makes RECQL5 helicase activity a novel target for chemotherapy.
Patterson K et al.
Oncotarget, 2016
RECQ5 helicase promotes resolution of conflicts between replication and transcription in human cells.
Urban V et al.
The Journal of Cell Biology, 2016
RECQL5 plays co-operative and complementary roles with WRN syndrome helicase.
Popuri V et al.
Nucleic Acids Research, 2013
A variant of the breast cancer type 2 susceptibility protein (BRC) repeat is essential for the RECQL5 helicase to interact with RAD51 recombinase for genome stabilization.
Islam MN et al.
The Journal of Biological Chemistry, 2012
Recruitment and retention dynamics of RECQL5 at DNA double strand break sites.
Popuri V et al.
Dna Repair, 2012
Helicase-inactivating mutations as a basis for dominant negative phenotypes.
Wu Y et al.
Cell Cycle (georgetown, Tex.), 2010
RECQL5/Recql5 helicase regulates homologous recombination and suppresses tumor formation via disruption of Rad51 presynaptic filaments.
Hu Y et al.
Genes & Development, 2007
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