Associated Papers
Gain-of-function mutations in RPA1 cause a syndrome with short telomeres and somatic genetic rescue.
Sharma R et al.
Blood, 2022
The helicase domain of Polθ counteracts RPA to promote alt-NHEJ.
Mateos-Gomez PA et al.
Nature Structural & Molecular Biology, 2017
RPA prevents G-rich structure formation at lagging-strand telomeres to allow maintenance of chromosome ends.
Audry J et al.
The Embo Journal, 2015
A novel allele of fission yeast rad11 that causes defects in DNA repair and telomere length regulation.
Ono Y et al.
Nucleic Acids Research, 2003
Characterization of the hyperrecombination phenotype of the pol3-t mutation of Saccharomyces cerevisiae.
Galli A et al.
Genetics, 2003
[Roles of ATM in DNA damage checkpoint and telomere maintenance].
Kanoh J et al.
Tanpakushitsu Kakusan Koso. Protein, Nucleic Acid, Enzyme, 2001
Rsp5, a ubiquitin-protein ligase, is involved in degradation of the single-stranded-DNA binding protein rfa1 in Saccharomyces cerevisiae.
Erdeniz N et al.
Molecular and Cellular Biology, 2000
Characterization of genetic interactions with RFA1: the role of RPA in DNA replication and telomere maintenance.
Smith J et al.
Biochimie, 2000
An allele of RFA1 suppresses RAD52-dependent double-strand break repair in Saccharomyces cerevisiae.
Smith J et al.
Genetics, 1999
Replication protein A (RPA): the eukaryotic SSB.
Iftode C et al.
Critical Reviews in Biochemistry and Molecular Biology, 1999
A novel allele of Saccharomyces cerevisiae RFA1 that is deficient in recombination and repair and suppressible by RAD52.
Firmenich AA et al.
Molecular and Cellular Biology, 1995
A mutation in the gene encoding the Saccharomyces cerevisiae single-stranded DNA-binding protein Rfa1 stimulates a RAD52-independent pathway for direct-repeat recombination.
Smith J et al.
Molecular and Cellular Biology, 1995
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