Associated Papers
Genomic and Reverse Translational Analysis Discloses a Role for Small GTPase RhoA Signaling in the Pathogenesis of Schizophrenia: Rho-Kinase as a Novel Drug Target.
Tanaka R et al.
International Journal of Molecular Sciences, 2023
Z-flipon variants reveal the many roles of Z-DNA and Z-RNA in health and disease.
Umerenkov D et al.
Life Science Alliance, 2023
37164635 (PubMed )
Supplement: LSA-2023-01962_TableS6.xlsx › Experimental_by_OMIM
Tracking Neoantigens by Personalized Circulating Tumor DNA Sequencing during Checkpoint Blockade Immunotherapy in Non-Small Cell Lung Cancer.
Jia Q et al.
Advanced Science (weinheim, Baden-wurttemberg, Germany), 2020
32382482 (PubMed )
Supplement: ADVS-7-1903410-s005.xlsx › P7
A novel rare variant R292H in RTN4R affects growth cone formation and possibly contributes to schizophrenia susceptibility.
Kimura H et al.
Translational Psychiatry, 2017
Genetic variants in Nogo receptor signaling pathways may be associated with early life adversity in schizophrenia susceptibility.
Andrews JL et al.
Bba Clinical, 2015
Genetic variants of Nogo-66 receptor with possible association to schizophrenia block myelin inhibition of axon growth.
Budel S et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 2008
Nogo Receptor 1 (RTN4R) as a candidate gene for schizophrenia: analysis using human and mouse genetic approaches.
Hsu R et al.
Plos One, 2007
Structural analysis of leucine-rich-repeat variants in proteins associated with human diseases.
Matsushima N et al.
Cellular and Molecular Life Sciences : Cmls, 2005
Mutations of the Nogo-66 receptor (RTN4R) gene in schizophrenia.
Sinibaldi L et al.
Human Mutation, 2004
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