Associated Papers
Proteomic subtypes enrich current acute myeloid leukemia nomenclature and reflect intrinsic pathogenesis alongside aging.
Cheng WY et al.
Blood, 2025
40829168 (PubMed )
Supplement: BLOOD_BLD-2024-027692-mmc5.xlsx › Sheet_1
Shwachman-Diamond syndromes: clinical, genetic, and biochemical insights from the rare variants.
Kawashima N et al.
Haematologica, 2023
Postazacitidine clone size predicts long-term outcome of patients with myelodysplastic syndromes and related myeloid neoplasms.
Nannya Y et al.
Blood Advances, 2023
36989067 (PubMed )
Supplement: BLOODA_ADV-2022-009564-mmc2.xlsx › SuppTable3
Validation and refinement of a RUNX1 mutation-associated gene expression signature in blast crisis chronic myeloid leukemia.
Lee KL et al.
Leukemia, 2022
35121847 (PubMed )
Supplement: 41375_2022_1508_MOESM1_ESM.xlsx › Supplementary_Table_5
Targeted next-generation sequencing identified novel mutations in triple-negative myeloproliferative neoplasms.
Chang YC et al.
Medical Oncology (northwood, London, England), 2017
Clonal evolution in relapsed acute myeloid leukaemia revealed by whole-genome sequencing.
Ding L et al.
Nature, 2012
22237025 (PubMed )
Supplement: NIHMS341298-supplement-1.pdf
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