Associated Papers
Remodeling of Ion Channel Trafficking and Cardiac Arrhythmias.
Blandin CE et al.
Cells, 2021
Sudden Cardiac Death-A New Insight Into Potentially Fatal Genetic Markers.
Primorac D et al.
Frontiers in Medicine, 2021
N-Glycosylation of the voltage-gated sodium channel β2 subunit is required for efficient trafficking of NaV1.5/β2 to the plasma membrane.
Cortada E et al.
The Journal of Biological Chemistry, 2019
Trafficking and Function of the Voltage-Gated Sodium Channel β2 Subunit.
Cortada E et al.
Biomolecules, 2019
Voltage-Gated Sodium Channel β Subunits and Their Related Diseases.
Bouza AA et al.
Handbook of Experimental Pharmacology, 2018
Trafficking and localisation to the plasma membrane of Nav 1.5 promoted by the β2 subunit is defective due to a β2 mutation associated with Brugada syndrome.
Dulsat G et al.
Biology of the Cell, 2017
Brugada syndrome: a fatal disease with complex genetic etiologies - still a long way to go.
Wu Y et al.
Forensic Sciences Research, 2017
Sodium channel β subunits: emerging targets in channelopathies.
O'Malley HA et al.
Annual Review of Physiology, 2015
Comprehensive Genetic Characterization of a Spanish Brugada Syndrome Cohort.
Selga E et al.
Plos One, 2015
A missense mutation in the sodium channel β2 subunit reveals SCN2B as a new candidate gene for Brugada syndrome.
Riuró H et al.
Human Mutation, 2013
The genetic component of Brugada syndrome.
Nielsen MW et al.
Frontiers in Physiology, 2013
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