Associated Papers
Identification of a novel Scn3b mutation in a Chinese Brugada syndrome pedigree: implications for Nav1.5 electrophysiological properties and intracellular distribution of Nav1.5 and Navβ3.
Fan J et al.
Frontiers in Cardiovascular Medicine, 2024
Two Novel Functional Mutations in Promoter Region of SCN3B Gene Associated with Atrial Fibrillation.
Lin L et al.
Life (basel, Switzerland), 2022
Molecular Pathology of Sodium Channel Beta-Subunit Variants.
Angsutararux P et al.
Frontiers in Pharmacology, 2021
Sodium channel β subunits: emerging targets in channelopathies.
O'Malley HA et al.
Annual Review of Physiology, 2015
Very early-onset lone atrial fibrillation patients have a high prevalence of rare variants in genes previously associated with atrial fibrillation.
Olesen MS et al.
Heart Rhythm, 2014
Channelopathies from mutations in the cardiac sodium channel protein complex.
Adsit GS et al.
Journal of Molecular and Cellular Cardiology, 2013
Novel SCN3B mutation associated with brugada syndrome affects intracellular trafficking and function of Nav1.5.
Ishikawa T et al.
Circulation Journal : Official Journal of the Japanese Circulation Society, 2013
Phenotypical manifestations of mutations in the genes encoding subunits of the cardiac sodium channel.
Wilde AA et al.
Circulation Research, 2011
Mutations in sodium channel β-subunit SCN3B are associated with early-onset lone atrial fibrillation.
Olesen MS et al.
Cardiovascular Research, 2011
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