Associated Papers
Predicting changes to INa from missense mutations in human SCN5A.
Clerx M et al.
Scientific Reports, 2018
Management of untreatable ventricular arrhythmias during pharmacologic challenges with sodium channel blockers for suspected Brugada syndrome.
Poli S et al.
Europace : European Pacing, Arrhythmias, and Cardiac Electrophysiology : Journal of the Working Groups on Cardiac Pacing, Arrhythmias, and Cardiac Cellular Electrophysiology of the European Society of Cardiology, 2018
Nadolol block of Nav1.5 does not explain its efficacy in the long QT syndrome.
Besana A et al.
Journal of Cardiovascular Pharmacology, 2012
Cardiac sodium channel dysfunction in sudden infant death syndrome.
Wang DW et al.
Circulation, 2007
Partial expression defect for the SCN5A missense mutation G1406R depends on splice variant background Q1077 and rescue by mexiletine.
Tan BH et al.
American Journal of Physiology. Heart and Circulatory Physiology, 2006
Tetrodotoxin-resistant Na+ channels in human neuroblastoma cells are encoded by new variants of Nav1.5/SCN5A.
Ou SW et al.
The European Journal of Neuroscience, 2005
A ubiquitous splice variant and a common polymorphism affect heterologous expression of recombinant human SCN5A heart sodium channels.
Makielski JC et al.
Circulation Research, 2003
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