Associated Papers
Clinical Features of Brugada Syndrome Patients With SCN5A Variants.
Okamura S et al.
Journal of Cardiovascular Electrophysiology, 2025
Genetic basis of sudden death after COVID-19 vaccination in Thailand.
Ittiwut C et al.
Heart Rhythm, 2022
Endophenotype effect sizes support variant pathogenicity in monogenic disease susceptibility genes.
Halford JL et al.
Nature Communications, 2022
36042188 (PubMed )
Supplement: 41467_2022_32009_MOESM4_ESM.xlsx › Supplementary_Data_2
Plasma MicroRNAs as noninvasive diagnostic biomarkers in patients with Brugada syndrome.
Ikeuchi Y et al.
Plos One, 2022
Genetic basis of cardiomyopathy and the genotypes involved in prognosis and left ventricular reverse remodeling.
Tobita T et al.
Scientific Reports, 2018
29386531 (PubMed )
Supplement: 41598_2018_20114_MOESM1_ESM.pdf
H558R, a common SCN5A polymorphism, modifies the clinical phenotype of Brugada syndrome by modulating DNA methylation of SCN5A promoters.
Matsumura H et al.
Journal of Biomedical Science, 2017
Common Variant Near HEY2 Has a Protective Effect on Ventricular Fibrillation Occurrence in Brugada Syndrome by Regulating the Repolarization Current.
Nakano Y et al.
Circulation. Arrhythmia and Electrophysiology, 2016
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