Associated Papers
Protein Quality Control of NKCC2 in Bartter Syndrome and Blood Pressure Regulation.
Laghmani K
Cells, 2024
Diacidic Motifs in the Carboxyl Terminus Are Required for ER Exit and Translocation to the Plasma Membrane of NKCC2.
Bakhos-Douaihy D et al.
International Journal of Molecular Sciences, 2022
Golgi Alpha1,2-Mannosidase IA Promotes Efficient Endoplasmic Reticulum-Associated Degradation of NKCC2.
Demaretz S et al.
Cells, 2021
New insights into the role of endoplasmic reticulum-associated degradation in Bartter Syndrome Type 1.
Shaukat I et al.
Human Mutation, 2021
Differential Effects of STCH and Stress-Inducible Hsp70 on the Stability and Maturation of NKCC2.
Bakhos-Douaihy D et al.
International Journal of Molecular Sciences, 2021
Genetic heterogeneity in patients with Bartter syndrome type 1.
Sun M et al.
Molecular Medicine Reports, 2017
Multiple evolutionarily conserved Di-leucine like motifs in the carboxyl terminus control the anterograde trafficking of NKCC2.
Zaarour N et al.
The Journal of Biological Chemistry, 2012
A highly conserved motif at the COOH terminus dictates endoplasmic reticulum exit and cell surface expression of NKCC2.
Zaarour N et al.
The Journal of Biological Chemistry, 2009
Mutations in the human Na-K-2Cl cotransporter (NKCC2) identified in Bartter syndrome type I consistently result in nonfunctional transporters.
Starremans PG et al.
Journal of the American Society of Nephrology : Jasn, 2003
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