Associated Papers
Impact of Zinc Transport Mechanisms on Embryonic and Brain Development.
Willekens J et al.
Nutrients, 2022
A common genetic variant in zinc transporter ZnT2 (Thr288Ser) is present in women with low milk volume and alters lysosome function and cell energetics.
Rivera OC et al.
American Journal of Physiology. Cell Physiology, 2020
Milk-derived miRNA profiles elucidate molecular pathways that underlie breast dysfunction in women with common genetic variants in SLC30A2.
Kelleher SL et al.
Scientific Reports, 2019
A genetic variant in SLC30A2 causes breast dysfunction during lactation by inducing ER stress, oxidative stress and epithelial barrier defects.
Lee S et al.
Scientific Reports, 2018
Exome Sequencing of SLC30A2 Identifies Novel Loss- and Gain-of-Function Variants Associated with Breast Cell Dysfunction.
Alam S et al.
Journal of Mammary Gland Biology and Neoplasia, 2015
Loss-of-function SLC30A2 mutants are associated with gut dysbiosis and alterations in intestinal gene expression in preterm infants.
Kelleher SL et al.
Gut Microbes, 0
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