Varaico: Literature-Extracted Genetic Variants

Gene:SLC30A2
Variant:p.G87R
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SLC30A2

NM_001004434.3:p.G87R

Associated Papers

Genetic disease risks of under-represented founder populations in New York City.

Isshiki M et al.

Plos Genetics, 2025

40554605 (PubMed )
Supplement: pgen.1011755.s006.xlsx › TableS2

Genetic disease risks of under-represented founder populations in New York City.

Isshiki M et al.

Medrxiv : the Preprint Server for Health Sciences, 2024

39399040 (PubMed )
Supplement: media-1.xlsx › TableS2

Haploinsufficiency underlies the neurodevelopmental consequences of SLC6A1 variants.

Silva DB et al.

American Journal of Human Genetics, 2024

38781976 (PubMed )
Supplement: mmc10.pdf

Impact of Zinc Transport Mechanisms on Embryonic and Brain Development.

Willekens J et al.

Nutrients, 2022

A novel homozygous mutation p.E88K in maternal SLC30A2 gene as a cause of transient neonatal zinc deficiency.

Li Z et al.

Experimental Dermatology, 2020

High proportion of transient neonatal zinc deficiency causing alleles in the general population.

Golan Y et al.

Journal of Cellular and Molecular Medicine, 2019

Alterations in ZnT1 expression and function lead to impaired intracellular zinc homeostasis in cancer.

Lehvy AI et al.

Cell Death Discovery, 2019

The role of the zinc transporter SLC30A2/ZnT2 in transient neonatal zinc deficiency.

Golan Y et al.

Metallomics : Integrated Biometal Science, 2017

Transient Neonatal Zinc Deficiency Caused by a Novel Mutation in the SLC30A2 Gene.

Liew HM et al.

Pediatric Dermatology, 2017

Novel mutations in SLC30A2 involved in the pathogenesis of transient neonatal zinc deficiency.

Itsumura N et al.

Pediatric Research, 2016

Molecular Basis of Transient Neonatal Zinc Deficiency: NOVEL ZnT2 MUTATIONS DISRUPTING ZINC BINDING AND PERMEATION.

Golan Y et al.

The Journal of Biological Chemistry, 2016

Exome Sequencing of SLC30A2 Identifies Novel Loss- and Gain-of-Function Variants Associated with Breast Cell Dysfunction.

Alam S et al.

Journal of Mammary Gland Biology and Neoplasia, 2015

Altered expression of two zinc transporters, SLC30A5 and SLC30A6, underlies a mammary gland disorder of reduced zinc secretion into milk.

Kumar L et al.

Genes & Nutrition, 2015

GESPA: classifying nsSNPs to predict disease association.

Khurana JK et al.

Bmc Bioinformatics, 2015

26206375 (PubMed )
Supplement: 12859_2015_673_MOESM2_ESM.xls › Sheet1

Genetic causes and gene–nutrient interactions in mammalian zinc deficiencies: acrodermatitis enteropathica and transient neonatal zinc deficiency as examples.

Kasana S et al.

Journal of Trace Elements in Medicine and Biology : Organ of the Society for Minerals and Trace Elements (gms), 2015

Current understanding of ZIP and ZnT zinc transporters in human health and diseases.

Kambe T et al.

Cellular and Molecular Life Sciences : Cmls, 2014

Molecular pathogenesis of spondylocheirodysplastic Ehlers-Danlos syndrome caused by mutant ZIP13 proteins.

Bin BH et al.

Embo Molecular Medicine, 2014

In situ dimerization of multiple wild type and mutant zinc transporters in live cells using bimolecular fluorescence complementation.

Lasry I et al.

The Journal of Biological Chemistry, 2014

Compound heterozygous mutations in SLC30A2/ZnT2 results in low milk zinc concentrations: a novel mechanism for zinc deficiency in a breast-fed infant.

Itsumura N et al.

Plos One, 2013

Transient Neonatal Zinc Deficiency Caused by a Heterozygous G87R Mutation in the Zinc Transporter ZnT-2 (SLC30A2) Gene in the Mother Highlighting the Importance of Zn (2+) for Normal Growth and Development.

Miletta MC et al.

International Journal of Endocrinology, 2013

A dominant negative heterozygous G87R mutation in the zinc transporter, ZnT-2 (SLC30A2), results in transient neonatal zinc deficiency.

Lasry I et al.

The Journal of Biological Chemistry, 2012

Transient neonatal zinc deficiency due to a new autosomal dominant mutation in gene SLC30A2 (ZnT-2).

Lova Navarro M et al.

Pediatric Dermatology, 0

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