Associated Papers
A novel homozygous mutation p.E88K in maternal SLC30A2 gene as a cause of transient neonatal zinc deficiency.
Li Z et al.
Experimental Dermatology, 2020
High proportion of transient neonatal zinc deficiency causing alleles in the general population.
Golan Y et al.
Journal of Cellular and Molecular Medicine, 2019
Novel mutations in SLC30A2 involved in the pathogenesis of transient neonatal zinc deficiency.
Itsumura N et al.
Pediatric Research, 2016
Genetic causes and gene–nutrient interactions in mammalian zinc deficiencies: acrodermatitis enteropathica and transient neonatal zinc deficiency as examples.
Kasana S et al.
Journal of Trace Elements in Medicine and Biology : Organ of the Society for Minerals and Trace Elements (gms), 2015
Compound heterozygous mutations in SLC30A2/ZnT2 results in low milk zinc concentrations: a novel mechanism for zinc deficiency in a breast-fed infant.
Itsumura N et al.
Plos One, 2013
Polymorphisms of SLC30A2 and selected perinatal factors associated with low milk zinc in Chinese breastfeeding women.
Qian L et al.
Early Human Development, 2012
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