Associated Papers
Characteristics and pharmacological responsiveness in hiPSC models of inherited cardiomyopathy.
Gerritse M et al.
Pharmacology & Therapeutics, 2025
Cryo-EM structure of the folded-back state of human β-cardiac myosin.
Grinzato A et al.
Biorxiv : the Preprint Server for Biology, 2023
Hereditary heart disease: pathophysiology, clinical presentation, and animal models of HCM, RCM, and DCM associated with mutations in cardiac myosin light chains.
Yadav S et al.
Pflugers Archiv : European Journal of Physiology, 2019
New Noonan syndrome model mice with RIT1 mutation exhibit cardiac hypertrophy and susceptibility to β-adrenergic stimulation-induced cardiac fibrosis.
Takahara S et al.
Ebiomedicine, 2019
30898653 (PubMed )
Supplement: mmc1.docx
Single cardiac ventricular myosins are autonomous motors.
Wang Y et al.
Open Biology, 2018
Remodeling of the heart in hypertrophy in animal models with myosin essential light chain mutations.
Kazmierczak K et al.
Frontiers in Physiology, 2014
Discrete effects of A57G-myosin essential light chain mutation associated with familial hypertrophic cardiomyopathy.
Kazmierczak K et al.
American Journal of Physiology. Heart and Circulatory Physiology, 2013
Mutations of ventricular essential myosin light chain disturb myosin binding and sarcomeric sorting.
Lossie J et al.
Cardiovascular Research, 2012
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