Associated Papers
The ribonuclease domain function is dispensable for SLFN11 to mediate cell fate decision during replication stress response.
Qi F et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms, 2023
Structural, molecular, and functional insights into Schlafen proteins.
Jo U et al.
Experimental & Molecular Medicine, 2022
SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells.
Okamoto Y et al.
Blood, 2021
SLFN11 Blocks Stressed Replication Forks Independently of ATR.
Murai J et al.
Molecular Cell, 2018
SLFN11 inhibits checkpoint maintenance and homologous recombination repair.
Mu Y et al.
Embo Reports, 2016
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