Associated Papers
A novel mouse model reproducing frontal alterations related to the prodromal stage of dementia with LEWY bodies.
Schueller E et al.
Neurobiology of Disease, 2024
The structural heterogeneity of α-synuclein is governed by several distinct subpopulations with interconversion times slower than milliseconds.
Chen J et al.
Structure (london, England : 1993), 2021
Treadmill exercise intervention improves gait and postural control in alpha-synuclein mouse models without inducing cerebral autophagy.
Minakaki G et al.
Behavioural Brain Research, 2019
Neuronal expression of familial Parkinson's disease A53T α-synuclein causes early motor impairment, reduced anxiety and potential sleep disturbances in mice.
Rothman SM et al.
Journal of Parkinson's Disease, 2013
Genetically engineered mouse models of Parkinson's disease.
Crabtree DM et al.
Brain Research Bulletin, 2012
Phenylbutyrate up-regulates the DJ-1 protein and protects neurons in cell culture and in animal models of Parkinson disease.
Zhou W et al.
The Journal of Biological Chemistry, 2011
Mouse models of cognitive deficits due to alpha-synuclein pathology.
Magen I et al.
Journal of Parkinson's Disease, 2011
Transgenic mice overexpressing tyrosine-to-cysteine mutant human alpha-synuclein: a progressive neurodegenerative model of diffuse Lewy body disease.
Zhou W et al.
The Journal of Biological Chemistry, 2008
Cysteine misincorporation in bacterially expressed human alpha-synuclein.
Masuda M et al.
Febs Letters, 2006
Tyrosine-to-cysteine modification of human alpha-synuclein enhances protein aggregation and cellular toxicity.
Zhou W et al.
The Journal of Biological Chemistry, 2004
[Modelling synucleinopathies in genetically modified animals--successes and failures].
Ninkina NN et al.
Molekuliarnaia Biologiia, 0
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