Associated Papers
Development of an aggregate-selective, human-derived α-synuclein antibody BIIB054 that ameliorates disease phenotypes in Parkinson's disease models.
Weihofen A et al.
Neurobiology of Disease, 2019
A pH-dependent switch promotes β-synuclein fibril formation via glutamate residues.
Moriarty GM et al.
The Journal of Biological Chemistry, 2017
Differences in the binding of copper(I) to α- and β-synuclein.
De Ricco R et al.
Inorganic Chemistry, 2015
Novel A18T and pA29S substitutions in α-synuclein may be associated with sporadic Parkinson's disease.
Hoffman-Zacharska D et al.
Parkinsonism & Related Disorders, 2013
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