Associated Papers
Human STAT3 variants underlie autosomal dominant hyper-IgE syndrome by negative dominance.
Asano T et al.
The Journal of Experimental Medicine, 2021
Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential.
Meixner I et al.
Orphanet Journal of Rare Diseases, 2020
A Novel STAT3 Mutation in a Qatari Patient With Hyper-IgE Syndrome.
Chaimowitz NS et al.
Frontiers in Pediatrics, 2019
The Ying and Yang of STAT3 in Human Disease.
Vogel TP et al.
Journal of Clinical Immunology, 2015
Heterozygous signal transducer and activator of transcription 3 mutations in hyper-IgE syndrome result in altered B-cell maturation.
Meyer-Bahlburg A et al.
The Journal of Allergy and Clinical Immunology, 2012
Defects along the T(H)17 differentiation pathway underlie genetically distinct forms of the hyper IgE syndrome.
Al Khatib S et al.
The Journal of Allergy and Clinical Immunology, 2009
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