Associated Papers
Aberrant function of pathogenic STAT3 mutant proteins is linked to altered stability of monomers and homodimers.
Kasembeli MM et al.
Blood, 2023
Human STAT3 variants underlie autosomal dominant hyper-IgE syndrome by negative dominance.
Asano T et al.
The Journal of Experimental Medicine, 2021
Aspergillus fumigatus Infection in Humans With STAT3-Deficiency Is Associated With Defective Interferon-Gamma and Th17 Responses.
Danion F et al.
Frontiers in Immunology, 2020
Structural Implications of STAT3 and STAT5 SH2 Domain Mutations.
de Araujo ED et al.
Cancers, 2019
A deep intronic splice mutation of STAT3 underlies hyper IgE syndrome by negative dominance.
Khourieh J et al.
Proceedings of the National Academy of Sciences of the United States of America, 2019
Hyper IgE recurrent infection syndrome in South Asia: is there a different outcome?
de Silva R et al.
Allergy, Asthma, and Clinical Immunology : Official Journal of the Canadian Society of Allergy and Clinical Immunology, 2018
Protein stabilization improves STAT3 function in autosomal dominant hyper-IgE syndrome.
Bocchini CE et al.
Blood, 2016
Blood CD4+CD45RO+CXCR5+ T cells are decreased but partially functional in signal transducer and activator of transcription 3 deficiency.
Mazerolles F et al.
The Journal of Allergy and Clinical Immunology, 2013
Autosomal dominant STAT3 deficiency and hyper-IgE syndrome: molecular, cellular, and clinical features from a French national survey.
Chandesris MO et al.
Medicine, 2012
Frequent and widespread vascular abnormalities in human signal transducer and activator of transcription 3 deficiency.
Chandesris MO et al.
Circulation. Cardiovascular Genetics, 2012
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