Associated Papers
Hyperactive STAT5 hijacks T cell receptor signaling and drives immature T cell acute lymphoblastic leukemia.
Suske T et al.
The Journal of Clinical Investigation, 2024
The neonatal microenvironment programs innate γδ T cells through the transcription factor STAT5.
Kadekar D et al.
The Journal of Clinical Investigation, 2020
STAT5A and STAT5B-Twins with Different Personalities in Hematopoiesis and Leukemia.
Maurer B et al.
Cancers, 2019
Src family kinases interfere with dimerization of STAT5A through a phosphotyrosine-SH2 domain interaction.
Fahrenkamp D et al.
Cell Communication and Signaling : Ccs, 2015
Targeting oncogenic interleukin-7 receptor signalling with N-acetylcysteine in T cell acute lymphoblastic leukaemia.
Mansour MR et al.
British Journal of Haematology, 2015
Serine phosphorylation of the Stat5a C-terminus is a driving force for transformation.
Friedbichler K et al.
Frontiers in Bioscience (landmark Edition), 2011
Productive parvovirus B19 infection of primary human erythroid progenitor cells at hypoxia is regulated by STAT5A and MEK signaling but not HIFα.
Chen AY et al.
Plos Pathogens, 2011
Genomic structure, expression and characterization of a STAT5 homologue from pufferfish (Tetraodon fluviatilis).
Sung SC et al.
European Journal of Biochemistry, 2003
Constitutive activation of STAT5 by a point mutation in the SH2 domain.
Ariyoshi K et al.
The Journal of Biological Chemistry, 2000
Constitutively active STAT5A and STAT5B in vitro and in vivo: mutation of STAT5 is not a frequent cause of leukemogenesis.
Yamada K et al.
International Journal of Hematology, 2000
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