Associated Papers
Single-cell sequencing reveals shared clonal signatures in nonmalignant B and tumor cells in T-prolymphocytic leukemia.
Hesselager C et al.
Blood Neoplasia, 2025
The structural influence of the oncogenic driver mutation N642H in the STAT5B SH2 domain.
Haas-Neill L et al.
Protein Science : a Publication of the Protein Society, 2025
T-cell prolymphocytic leukemia, a case report and review of the literature.
GONZáLEZ-RODRíGUEZ LM et al.
Oncology Research, 2025
JAK/STAT in human diseases: a common axis in immunodeficiencies and hematological disorders.
Djidjik R et al.
Frontiers in Immunology, 2025
STAT5B mutations in myeloid neoplasms differ by disease subtypes but characterize a subset of chronic myeloid neoplasms with eosinophilia and/or basophilia.
Yin CC et al.
Haematologica, 2024
Monomorphic epitheliotropic intestinal T-cell lymphoma comprises morphologic and genomic heterogeneity impacting outcome.
Veloza L et al.
Haematologica, 2023
Targeted panel sequencing in the routine diagnosis of mature T- and NK-cell lymphomas: report of 128 cases from two German reference centers.
Böck J et al.
Frontiers in Oncology, 2023
Defining TCRγδ lymphoproliferative disorders by combined immunophenotypic and molecular evaluation.
Teramo A et al.
Nature Communications, 2022
Identification of novel STAT5B mutations and characterization of TCRβ signatures in CD4+ T-cell large granular lymphocyte leukemia.
Bhattacharya D et al.
Blood Cancer Journal, 2022
STAT5b: A master regulator of key biological pathways.
Smith MR et al.
Frontiers in Immunology, 2022
STAT3 and STAT5B mutations have unique distribution in T-cell large granular lymphocyte proliferations and advanced myeloid neoplasms.
Qu S et al.
Leukemia & Lymphoma, 2021
JAK/STAT-Activating Genomic Alterations Are a Hallmark of T-PLL.
Wahnschaffe L et al.
Cancers, 2019
Twins with different personalities: STAT5B-but not STAT5A-has a key role in BCR/ABL-induced leukemia.
Kollmann S et al.
Leukemia, 2019
Nonclassical GH Insensitivity: Characterization of Mild Abnormalities of GH Action.
Storr HL et al.
Endocrine Reviews, 2019
Enteropathy-associated T cell lymphoma subtypes are characterized by loss of function of SETD2.
Moffitt AB et al.
The Journal of Experimental Medicine, 2017
The Genetic Basis of Hepatosplenic T-cell Lymphoma.
McKinney M et al.
Cancer Discovery, 2017
High incidence of activating STAT5B mutations in CD4-positive T-cell large granular lymphocyte leukemia.
Andersson EI et al.
Blood, 2016
Type II enteropathy-associated T-cell lymphoma features a unique genomic profile with highly recurrent SETD2 alterations.
Roberti A et al.
Nature Communications, 2016
Genes encoding members of the JAK-STAT pathway or epigenetic regulators are recurrently mutated in T-cell prolymphocytic leukaemia.
López C et al.
British Journal of Haematology, 2016
Genomic analyses reveal recurrent mutations in epigenetic modifiers and the JAK-STAT pathway in Sézary syndrome.
Kiel MJ et al.
Nature Communications, 2015
Integrated genomic sequencing reveals mutational landscape of T-cell prolymphocytic leukemia.
Kiel MJ et al.
Blood, 2014