Associated Papers
STAT5B leukemic mutations, altering SH2 tyrosine 665, have opposing impacts on immune gene programs.
Lee HK et al.
Life Science Alliance, 2025
Disease-Associated Mutations of the STAT5B SH2 Domain Regulate Cytokine-Driven Enhancer Function and Mammary Development.
Lee HK et al.
Journal of Mammary Gland Biology and Neoplasia, 2025
Maintenance of hematopoietic stem cells by tyrosine-unphosphorylated STAT5 and JAK inhibition.
Williams MJ et al.
Blood Advances, 2025
STAT5B leukemic mutations, altering SH2 tyrosine 665, have opposing impacts on immune gene programs.
Lee HK et al.
Biorxiv : the Preprint Server for Biology, 2024
Tyrosine phosphorylation of both STAT5A and STAT5B is necessary for maximal IL-2 signaling and T cell proliferation.
Lin JX et al.
Nature Communications, 2024
The Derlin-1-Stat5b axis maintains homeostasis of adult hippocampal neurogenesis.
Murao N et al.
Embo Reports, 2024
STAT5A and STAT5B-Twins with Different Personalities in Hematopoiesis and Leukemia.
Maurer B et al.
Cancers, 2019
Biochemical characterization of diverse Stat5b-binding enhancers that mediate growth hormone-activated insulin-like growth factor-I gene transcription.
Varco-Merth B et al.
Plos One, 2012
Modulation of signal transducer and activator of transcription 5b activity in breast cancer cells by mutation of tyrosines within the transactivation domain.
Weaver AM et al.
Molecular Endocrinology (baltimore, Md.), 2006
Serine phosphorylation of GH-activated signal transducer and activator of transcription 5a (STAT5a) and STAT5b: impact on STAT5 transcriptional activity.
Park SH et al.
Molecular Endocrinology (baltimore, Md.), 2001
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