Associated Papers
Mice deficient in the C-terminal domain of TAR DNA-binding protein 43 develop age-dependent motor dysfunction associated with impaired Notch1-Akt signaling pathway.
Nishino K et al.
Acta Neuropathologica Communications, 2019
Transgenic and physiological mouse models give insights into different aspects of amyotrophic lateral sclerosis.
De Giorgio F et al.
Disease Models & Mechanisms, 2019
A nonsense mutation in mouse Tardbp affects TDP43 alternative splicing activity and causes limb-clasping and body tone defects.
Ricketts T et al.
Plos One, 2014
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