Associated Papers
Hierarchical deployment of Tbx3 dictates the identity of hypothalamic KNDy neurons to control puberty onset.
Shi X et al.
Science Advances, 2022
The roles and regulation of TBX3 in development and disease.
Khan SF et al.
Gene, 2020
Hypogonadotropic hypogonadism and pituitary hypoplasia as recurrent features in Ulnar-Mammary syndrome.
Galazzi E et al.
Endocrine Connections, 2018
30550377 (PubMed )
Supplement: supplementary_table_3.pdf
GESPA: classifying nsSNPs to predict disease association.
Khurana JK et al.
Bmc Bioinformatics, 2015
26206375 (PubMed )
Supplement: 12859_2015_673_MOESM2_ESM.xls › Sheet1
Tbx3 is a downstream target of the Wnt/beta-catenin pathway and a critical mediator of beta-catenin survival functions in liver cancer.
Renard CA et al.
Cancer Research, 2007
The T-box repressors TBX2 and TBX3 specifically regulate the tumor suppressor gene p14ARF via a variant T-site in the initiator.
Lingbeek ME et al.
The Journal of Biological Chemistry, 2002
Structure of the DNA-bound T-box domain of human TBX3, a transcription factor responsible for ulnar-mammary syndrome.
Coll M et al.
Structure (london, England : 1993), 2002
TBX-3, the gene mutated in Ulnar-Mammary Syndrome, is a negative regulator of p19ARF and inhibits senescence.
Brummelkamp TR et al.
The Journal of Biological Chemistry, 2002
The spectrum of mutations in TBX3: Genotype/Phenotype relationship in ulnar-mammary syndrome.
Bamshad M et al.
American Journal of Human Genetics, 1999
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