Associated Papers
Anastomotic leak: genetic aspects of prediction and choice of surgical treatment tactics.
Usenko OY et al.
Wiadomosci Lekarskie (warsaw, Poland : 1960), 2024
The polymorphisms of extracellular matrix-remodeling genes are associated with pelvic organ prolapse.
Li L et al.
International Urogynecology Journal, 2022
34973089 (PubMed )
Supplement: 192_2021_4917_MOESM1_ESM.xls › Sheet1
ANALYSIS OF POLYMORPHISM OF MATRIX METALLOPROTEINASE-2 (C-1306 → T) AND TISSUE INHIBITORS OF METALLOPROTEINASE-2 (G303 → A) GENES IN PATIENTS WITH ANASTOMOTIC LEAK IN HOLLOW DIGESTIVE ORGANS.
Voitiv Y et al.
Georgian Medical News, 2020
A protective polymorphism in MMP16, improved blood gas levels, and chronic obstructive pulmonary diseases: Family and two population-based studies.
Zhang Z et al.
Human Mutation, 2020
Tissue Inhibitor of Metalloproteinase-2 Polymorphisms and Risk for HIV-Associated Neurocognitive Disorder.
Singh H et al.
Mediators of Inflammation, 2019
Association of TIMP-2-418G/C and TIMP-2-303G/A with gastric cancer: a meta-analysis.
Yang Y et al.
Oncotargets and Therapy, 2016
TBX1 mutation identified by exome sequencing in a Japanese family with 22q11.2 deletion syndrome-like craniofacial features and hypocalcemia.
Ogata T et al.
Plos One, 2014
24637876 (PubMed )
Supplement: pone.0091598.s004.pdf
TIMP-2 gene polymorphism is associated with intracerebral hemorrhage.
Reuter B et al.
Cerebrovascular Diseases (basel, Switzerland), 2009
[Analysis of tissue inhibitor of metalloproteinase-2 gene polymorphisms in a caucasian population with abdominal aortic aneurysms].
Hinterseher I et al.
Zentralblatt Fur Chirurgie, 2008
Role of matrix metalloproteinase, tissue inhibitor of metalloproteinase and tumor necrosis factor-alpha single nucleotide gene polymorphisms in inflammatory bowel disease.
Meijer MJ et al.
World Journal of Gastroenterology, 2007
Tissue inhibitor of metalloproteinases-1, -2, and -3 polymorphisms in a white population with intracranial aneurysms.
Krex D et al.
Stroke, 2003
Highly multiplexed genotyping of coronary artery disease-associated SNPs using MALDI-TOF mass spectrometry.
Nakai K et al.
Human Mutation, 2002
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