Associated Papers
Aggressive Squamous Cell Carcinoma in a Case of Epidermodysplasia Verruciformis Carrying a TMC6 Splice-site Mutation.
Yokoi K et al.
Acta Dermato-venereologica, 2023
Identification and Splicing Characterization of Novel TMC6 and TMC8 Variants Associated With Epidermodysplasia Verruciformis in Three Chinese Families.
Wang R et al.
Frontiers in Genetics, 2021
34386043 (PubMed )
Supplement: Data_Sheet_1.docx
Human knockouts and phenotypic analysis in a cohort with a high rate of consanguinity.
Saleheen D et al.
Nature, 2017
28406212 (PubMed )
Supplement: NIHMS857566-supplement-Suppl__Table_1.xlsx › Supplementary_Table_1
The EVER genes - the genetic etiology of carcinogenesis in epidermodysplasia verruciformis and a possible role in non-epidermodysplasia verruciformis patients.
Kalińska-Bienias A et al.
Postepy Dermatologii I Alergologii, 2016
Epidermodysplasia verruciformis.
Burger B et al.
Current Problems in Dermatology, 2014
Regulation of cellular zinc balance as a potential mechanism of EVER-mediated protection against pathogenesis by cutaneous oncogenic human papillomaviruses.
Lazarczyk M et al.
The Journal of Experimental Medicine, 2008
Four mutations in Epidermodysplasia verruciformis 1 (EVER1) gene are not contributors to susceptibility in RRP.
Donfack J et al.
International Journal of Pediatric Otorhinolaryngology, 2006
Mutations in two adjacent novel genes are associated with epidermodysplasia verruciformis.
Ramoz N et al.
Nature Genetics, 2002
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