Associated Papers
An ALS-associated mutant SOD1 protein can be eliminated in microglia culture by selective autophagy.
Murakami K et al.
Neuroscience, 2026
Dysbiosis and Neurodegeneration in ALS: Unraveling the Gut-Brain Axis.
Sharma VK
Neuromolecular Medicine, 2025
Massive transcriptome sequencing of human spinal cord tissues provides new insights into motor neuron degeneration in ALS.
D'Erchia AM et al.
Scientific Reports, 2017
RNAseq Analyses Identify Tumor Necrosis Factor-Mediated Inflammation as a Major Abnormality in ALS Spinal Cord.
Brohawn DG et al.
Plos One, 2016
27487029 (PubMed )
Supplement: pone.0160520.s017.xlsx › Supplementary_Table_8
Nrf2/ARE Signaling Pathway: Key Mediator in Oxidative Stress and Potential Therapeutic Target in ALS.
Petri S et al.
Neurology Research International, 2012
The Role of immune and inflammatory mechanisms in ALS.
McCombe PA et al.
Current Molecular Medicine, 2011
Proteomic characterization of lipid raft proteins in amyotrophic lateral sclerosis mouse spinal cord.
Zhai J et al.
The Febs Journal, 2009
Mutant copper-zinc superoxide dismutase associated with amyotrophic lateral sclerosis binds to adenine/uridine-rich stability elements in the vascular endothelial growth factor 3'-untranslated region.
Li X et al.
Journal of Neurochemistry, 2009
Analysis of genes critical for growth regulation identifies Insulin-like Growth Factor 2 Receptor variations with possible functional significance as risk factors for osteosarcoma.
Savage SA et al.
Cancer Epidemiology, Biomarkers & Prevention : a Publication of the American Association for Cancer Research, Cosponsored by the American Society of Preventive Oncology, 2007
Informatics-assisted protein profiling in a transgenic mouse model of amyotrophic lateral sclerosis.
Lukas TJ et al.
Molecular & Cellular Proteomics : Mcp, 2006
Disease mechanisms revealed by transcription profiling in SOD1-G93A transgenic mouse spinal cord.
Olsen MK et al.
Annals of Neurology, 2001
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