Associated Papers
SPOP-dependent destabilization of SYT12 in a GSK-3β-dependent manner in papillary thyroid cancer cells.
Zhou S et al.
Clinical and Experimental Medicine, 2025
TP53_PROF: a machine learning model to predict impact of missense mutations in TP53.
Ben-Cohen G et al.
Briefings in Bioinformatics, 2022
35043155 (PubMed )
Supplement: supplementary_table_s1_bbab524.xlsx › S1B._Negative_Set
Regulation of p53 by the 14-3-3 protein interaction network: new opportunities for drug discovery in cancer.
Falcicchio M et al.
Cell Death Discovery, 2020
Downregulation of the DNA Repair Gene DDB2 by Arecoline Is through p53's DNA-Binding Domain and Is Correlated with Poor Outcome of Head and Neck Cancer Patients with Betel Quid Consumption.
Wang YC et al.
Cancers, 2020
Reproducible Analysis of Post-Translational Modifications in Proteomes--Application to Human Mutations.
Holehouse AS et al.
Plos One, 2015
26659599 (PubMed )
Supplement: pone.0144692.s002.xlsx › suppTable_2.txt
TP53 mutations in human cancer: database reassessment and prospects for the next decade.
Leroy B et al.
Human Mutation, 2014
24665023 (PubMed )
Supplement: supplement_1.pdf
Potential conformational heterogeneity of p53 bound to S100B(ββ).
McDowell C et al.
Journal of Molecular Biology, 2013
Phosphorylation at carboxyl-terminal S373 and S375 residues and 14-3-3 binding are not required for mouse p53 function.
Lee MK et al.
Neoplasia (new York, N.y.), 2007
Role of phosphorylation in p53 acetylation and PAb421 epitope recognition in baculoviral and mammalian expressed proteins.
Warnock LJ et al.
The Febs Journal, 2005
Endoplasmic reticulum stress induces p53 cytoplasmic localization and prevents p53-dependent apoptosis by a pathway involving glycogen synthase kinase-3beta.
Qu L et al.
Genes & Development, 2004
The interaction of p53 with the nuclear matrix is mediated by F-actin and modulated by DNA damage.
Okorokov AL et al.
Oncogene, 2002
Inhibition of p53 transcriptional activity by the S100B calcium-binding protein.
Lin J et al.
The Journal of Biological Chemistry, 2001
Increased p53 phosphorylation after microtubule disruption is mediated in a microtubule inhibitor- and cell-specific manner.
Stewart ZA et al.
Oncogene, 2001
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