Associated Papers
TRAF6 Phosphorylation Prevents Its Autophagic Degradation and Re-Shapes LPS-Triggered Signaling Networks.
Busch J et al.
Cancers, 2021
34298830 (PubMed )
Supplement: cancers-13-03618-s001.zip › Supplementary_Files.pdf
Two Antagonistic MALT1 Auto-Cleavage Mechanisms Reveal a Role for TRAF6 to Unleash MALT1 Activation.
Ginster S et al.
Plos One, 2017
Presenilins are novel substrates for TRAF6-mediated ubiquitination.
Yan R et al.
Cellular Signalling, 2013
Pellino3 targets the IRF7 pathway and facilitates autoregulation of TLR3- and viral-induced expression of type I interferons.
Siednienko J et al.
Nature Immunology, 2012
Human interleukin-1 receptor-associated kinase-2 is essential for Toll-like receptor-mediated transcriptional and post-transcriptional regulation of tumor necrosis factor alpha.
Flannery SM et al.
The Journal of Biological Chemistry, 2011
TRAF6 is autoinhibited by an intramolecular interaction which is counteracted by trans-ubiquitination.
Wang KZ et al.
Journal of Cellular Biochemistry, 2010
Act1, a U-box E3 ubiquitin ligase for IL-17 signaling.
Liu C et al.
Science Signaling, 2009
E2 interaction and dimerization in the crystal structure of TRAF6.
Yin Q et al.
Nature Structural & Molecular Biology, 2009
Nuclear tumor necrosis factor receptor-associated factor 6 in lymphoid cells negatively regulates c-Myb-mediated transactivation through small ubiquitin-related modifier-1 modification.
Pham LV et al.
The Journal of Biological Chemistry, 2008
Site-specific Lys-63-linked tumor necrosis factor receptor-associated factor 6 auto-ubiquitination is a critical determinant of I kappa B kinase activation.
Lamothe B et al.
The Journal of Biological Chemistry, 2007
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